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A main pathogenic initiator of AD and related dementias is oxidative stress, which corrodes the brain, called oxidative damage. (Representational Image: Unsplash)
Medicine

New Study Reveals Why Defense Against Brain Corrosion Declines in Alzheimer Disease (AD) Patients

MBT Desk
2 min read
Findings offer hope for development of new medications
These Numbers Put 12 Rare Diseases on the Map (Representational Image: Unsplash)
Medicine

12 Rare Diseases Added to International Classification List, Bringing Recognition and Awareness

MBT Desk
3 min read
New codes in the ICD-10-CM will help researchers and clinicians track and study rare diseases, and could lead to improved diagnosis and treatment.
The image shows Alzheimer's disease risk pathways identified by UCLA researchers.
Medicine

New Study Maps Four Key Pathways to Alzheimer’s Disease

MBT Desk
2 min read
UCLA Health researchers identify sequential diagnostic patterns that could improve early detection and prevention strategies
Abnormal Tau, a Sign of Alzheimer’s Disease in the Brain, Is Present in Dying Nerve Cells in the Retina
MedBound Blog

Research Tip Sheet: Proteins in Eye’s Nerve Cells Linked to Alzheimer’s Disease

MBT Desk
2 min read
Abnormal Tau, a Sign of Alzheimer’s Disease in the Brain, Is Present in Dying Nerve Cells in the Retina and Linked to Cognitive Decline, Study Finds
Research shows that Herpes virus may contribute to Alzheimer's.
Medicine

Herpesviruses May Contribute to Alzheimer’s Disease via Transposable Elements

MBT Desk
3 min read
HSV-1 is associated with Alzheimer’s-associated transposable elements in infected, Alzheimer’s-affected brains, which may be reversed through available drugs.
FDA-approved drugs may be repurposed to target disease-associated microglia in Alzheimer’s disease without affecting the healthy type. (Image: Pexels)
Medicine

Network-Based Analyses Uncover How Neuroinflammation-Causing Microglia in Alzheimer’s Disease Form

MBT Desk
4 min read
Cleveland Clinic-led study strengthens the connection between changes in microglia and Alzheimer’s disease and identifies drugs that may stop or reverse these changes.
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